Posted with the permission of A.J. Lindeman dated March 25, 2007. Journal of Degenerative Diseases 2002 Brucella Abortus Bang By A. J. Lindeman INTRODUCTION
Until 1861 there was never a difference in the identity of the illness. MARSTON was the first to detail Brucellosis and gave it the name Mediterranean Fever or Gastric Intermittent Fever. Other synonyms for this illness are: Undulant fever; Mediterranean fever; Gastric fever; Malta fever; Rock fever; Gibraltar fever; Melitoccie Goat fever; Texas fever; Rio Grande fever; Brucella fever; Bang's fever. Brucellus Abortus (Bang) was first isolated and described as a bacillus by Bang in conjunction with Stribolt in 1897. They took the organism from the foetus and the foetal membrane of cows that had aborted, and isolated these organisms. Later it was determined that the cause was from the infected abortion of the cow. The illness is now known as the "Illness of Bang" or "Bang's Disease" in animals, and "Brucella Abortus Bang" in humans. In all animals that chew cud and in pigs this illness causes natural abortions and is transferable to man. ETIOLOGY
Brucella melitensis: (3 biotypes) is to be found in sheep and goats and is enzootic (present in an animal community at all times), but occurring in only small numbers of cases--a disease of low morbidity which is constantly present in an animal community in lands around the Mediterranean, South East Russia, Mongolia, the Middle East and Latin America. Sheep and goats may also be infected by Brucella Suis and Brucella Abortus. Brucella suis: (4 biotypes) is to be found in pigs and is enzootic in Latin America, sporadically in Europe, Asia, Africa and Oceania. Pigs may be infected by Brucella Abortus. Brucella neotoma is to be found in rats in the deserts of West America. Brucella ovis is found in sheep, and is spread over all areas where sheep are bred. Epididymitis in sheep is caused by Brucella Ovis. Brucella canis is found in cats and dogs and has been bacterially located in America, Brazil, Argentina, Mexico, Czechoslovakia, Germany, Japan and Madagascar. Dogs and cats may also be infected by Brucella Suis, Brucella Abortus, and Brucella Melitensis. Symptoms and Pathology
HUGHES differentiated Brucellosis into three main types: a. Malignant, b. Undulant, c. Changeable. He also recognized an ambulant type and an irregular mixed and chronic type. A. The Malignant Type Infections of this type as a result of Brucella Abortus or Brucella Suis seldom occur. They are recognised by a sudden onset of acute duration and is often fatal. In most cases the temperature is high and does not abate and has an extreme hyperpyrexia prior to death. Exhaustion, severe headache and backache, anorexia, constipation and stiffness is noticeable. At some stage a state of delirium and coma may occur. Perspiration is not excessive. The spleen will certainly be enlarged. B. The Undulant Type The most noticeable characteristic of this type is the appearance of the recidivation. If short intermittent periods of the apyrexia occur it creates the impression of a wave-like temperature. In the beginning people complain of tiredness, general pain, headaches and anorexia….. Sometimes this is confused with an influenza attack. The people are hardly over the one attack when it begins a new. By the second attack the symptoms have often intensified. Constipation and sleeplessness also occur. In the early stage nightly sweats can occur but it is more visible in later developmental phases. Characteristically the temperature rises daily until the maximum is reached. This development then declines in intensity and length of duration. C. The Changeable Type The beginning phase is deceptive. The patient feels progressively tired in the early afternoon. General pain, headaches, no desire to eat, becoming cold in the late afternoon, and sleeplessness occurs frequently. A fever is often suspected. Backache, stiffness, and pain in the neck and joints, constipation and weight loss are also symptoms. With some patients there is also a dry cough and severe nightly sweats. Physical inspection shows no abnormalities except symptoms of anaemia, weakness, anorexia, palpable spleen and a sensitive intestinal area. D. Ambulant Type The duration of the illness is short and mild. In the beginning it is deceptive and the-only constant symptoms are tiredness and a loss of energy. E. The Irregular, Mixed And Chronic Type When diagnosing Brucella we must always bear in mind Atypical forms. These infections can imitate other symptoms. The chronic form of this illness can display the following clinical manifestations: A wandering form of typhus-like fever; Tuberculosis; Bronchopneumonia; Meningitis; Cystitis; Rheumatism; Appendicitis; Orchitis; Gonorrhoeal Epididymitis. Chronic cases can display neither any symptoms nor any signs of this illness. During the first months of the illness the patient may complain of tiredness and pain in the back of the head. Pain in the joints and back often occur. Many of the patients tell of vague forms of gastritis and a pain in the right lower abdomen which looks like chronic appendicitis. Sometimes in the evening there is a rise in the temperature. Some patients develop neurological symptoms e.g. depressions, are easily angered, crying without a reason, sleeplessness and tremors. It sometimes occurs that the patient suffers from a temporary loss of memory, speech defects, photophobia and a loss of feeling in the fingers, toes, arms and legs. The chronic form of this illness can form a purely focal infection. The focus may be found in the tonsils, roots of the teeth, the gall bladder, bones, lymph glands, joints or genitals. More and more evidence shows that people immune to Brucellosis show an allergic reaction which is often confused with a chronic Brucellosis. Brucellosis In
Children 1. Weakness in the form of tiredness. 2. Perspiration-varies a lot in length of duration from short to long ... particularly after midnight and can be mild or severe. 3. Cold shivers. 4. Pain: Sometimes becoming more pronounced with exercise and movement and can be confused with influenza, stiff neck and shoulders, arthralgia, pains in the abdomen (sometimes with cramps) but in general the pains are definite and severe and sometimes last a long while but to locate the exact point of pain is very difficult. Sometimes it is found in the regions of the ovaria, epigastrium and appendix. This symptom has the danger that it can lead to an operation that it is not necessary. 5. Gastro-intestinal problems: Anorexia, billiousness and vomiting, and constipation are part and parcel of the intensity of the infection. 6. Respiratory Symptoms: Acute symptoms of the upper respiratory area appear at the beginning of the infection, dry cough, pharyngeal irritation, bronchopneumonia, miliary tuberculosis, pulmonary abscesses, and tonsillitis. 7. Neuropsychiatric symptoms: Marked by depression, tendency to sleepiness and intoxication, loss of memory, over sensitivity, crying without a reason, uncertain, shy, childish manner, shaking and tremors so that the patient cannot write or bring a cup to the lips without messing, unwilling to talk, aphonia, temporary loss of movement in the arms and legs. 8. Uro-genital: Cystitis, pyelitis, burning feeling when urinating. 9. Cardiovascular: Palpitations, irritated heart, dizziness, and other cardiovascular symptoms have an influence on complications. 10. Loss of weight: Progressive weight loss--anorexia. Localized Brucella
Infections It is important to note that people who have been vaccinated for cholera sometimes have a positive reaction to Brucellosis. (The vaccine is often cultured in cattle and unless care is taken by the veterinarian, this could be a Brucellosis infected cow used as a culture base). Brucella Abortus Bang may be confused with Salmonella, Malaria and Mononucleosis infections. Transmission A person is infected by contact with the animals or direct or indirect contact with the use of animal products e.g. fresh cheese, milk or meat. Brucella organisms are often killed in sour milk, sour cream, butter and fermented cheese which is more than three months old, but there is no guarantee. Brucella Abortus is also found in the areas of the North Pole as a result of the eating of bone marrow and raw meat of the reindeer and caribou which have been infected with Brucella Suis biotype 4. The organism is resistant to pickling and smoking and thus can be transferred to humans very easily. It is also possible that raw green vegetables and water may be infected by animal faeces or urine. This is a source of the infection, or where "organic farming" is practised, and the soil is fertilized using fresh dung or dung that has not been well rotted for several years. (A common fault in Organic Farming in the U.K.). Human Brucellosis (Brucella Abortus Bang) is mostly the result of direct contact with the foetas and foetal membranes, and/or the afterbirth of the calf, or with contact of vaginal secretions of the cow, faeces or carcasses of any infected animal. The organism enters humans via skin damage or via the mucous membranes including the conjunctiva. There are cases in which the Brucellosis has been transmitted via the breathing in of Brucellosis in aerosol form in an abattoir or laboratory. Humans are also susceptible to the Brucella Canis infection. Many of these cases are to be found amongst laboratory workers, kennel owners where the breeding of dogs takes place, and in an infected dog as a pet. A thing of much importance is that it is transferable via the sperm to the woman who when she falls pregnant is able to transfer this to the foetus. (We have proven up to 3 generations of transmission.) Brucella is able to reach all organs in the body because it is transmitted via the blood. It is a possible cause of many menstruation problems e.g. heavy menstruation, dysmenorrhea, amenorrhea, and even spontaneous abortions. (today we even do not rule out Brucellosis as a cause of Fibromas and Myoma of the Uterus). Worst of all we have now proven conclusively that Brucellosis is the vector for other infections such as Tuberculosis and at least 4 types of Mycoplasma infections. Brucella melitense
Where does it all come from? I record this section which follows merely as background knowledge for my studies:- At the end of the Second World War, Holland was liberated in the southern provinces by the American troops who had goat mascots with them. These goats were carriers of Brucellosis. The U.S.A. troops carried truckloads of food for these mascots with them, as Europe was devastated by the war. The goats were housed in the near empty stables of the Dutch farmers, who were given food for the mascots in plentiful supply. Much was used immediately on their own starving stock. The droppings of the goats were spread on the land to fertilise the soil. In the Provinces of Limburg and Brabant this was the order of the day. Little wild rabbits came to eat of these droppings. They were a very rich source of food which was already partially digested. Thus the entire wild rabbit population became carriers of Brucellosis. In turn the people in these provinces trapped the rabbits and ate them, and entire families became ill. Some died. A11 this is recorded in the medical annals of Holland in a book called the Codex Medicus. This in an official diary of the medical history of Holland. In those years Holland possessed a blood type culture system to detect the bacteria. It was a special device (that looked like an oven) in which blood was cultured for 19 days, at the blood temperature of the patient. It required a 24 hours a day monitoring system, using three shifts of personnel. One culture was costing the National Health DFL 2000 per culture. The average tradesman's salary was only DFL 75 per week (this was back in 1945 47). There was no cure for the disease. When there is an outbreak of Brucellosis, the Ministry of Health must be notified who in turn must notify the World Health Organisation, and then the government is obliged to stop all exports of dairy produce, bovine sperm, etc. In 1947, only two years after thousands of people had died in Holland of Brucellosis, the Dutch government made it known that it had stamped out Brucella Abortus Bang and that the milk was of such high quality that regular testing had become obsolete. The equipment was destroyed, and no further blood tests cultures for this disease have been done since. This is not the end of it. In 1990 Holland destroyed more than 1,130 cows for suspected Brucellosis; in 1991, more than 3,000 cows were destroyed, and in 1992, over a thousand were destroyed. You find the occasional newspaper article announcing the destruction of the cows and stables, followed by an immediate announcement from the Ministry of Health saying that it is all routine and that the infection has presented no threat to the human population. In the very next sentence it is stated that the meat is to be destroyed, and may not be eaten. The latest notice in the newspaper I have is 19/8/95 in the "Telegraaf", a nation wide newspaper in Holland, where the State Veterinarians had noticed this disease at various farms in the Barchem area (Eastern Holland). The article did not reveal how many cows the state destroyed at this juncture. However, in 1990 it was published by the State Health department in Holland that eating the meat of the Brucellosis infected cattle was not dangerous. It has become a standard joke amongst the population in that every time they find an outbreak of Brucellosis, the state veterinarians blame it on the Belgians allowing their cattle to stray over our borders or selling their diseased cattle to the Dutch farmers. Likewise the population is always bemused by the state's hastily added announcement stating that this outbreak has not in any way placed at risk the population who might have been in contact with these cattle or eaten the meat or other products from these cows. From my story you can deduce so far that:
In the USSR, or whatever you would like to now call this area, they have large but unofficial unreported Brucellosis in bovines, horses, pigs, reindeer, goats, sheep, dogs and many of the wild animals in the entire area; they also admit to a "Wild Hare Brucellosis (Brucellosis Suis Thomsen)" in the South-East of the Russian States or Satellite countries. The German researchers (East and West Germany) even report a contamination of the so called "British Melitensis" variant of the disease. (BORGER and SEELEMANN). They published a map of the various: German "States" showing in which "country (state)" the percentage of Brucellosis contamination was in 1958-59. One contamination spreader according to German researchers is the insect population, the fly. Stomoxys Calcitrans has been proved to be responsible for contaminating wild boars. (WELLMANN) and HARNACH admitted that the Musca Domestica fly was responsible for carrying the disease from cattle. He also offered the theory that mosquitoes actively carried the disease over to human beings. In this connection HARNACH cites a research into miscarriages in the stables that reported that 11% of the insects inspected were carriers of live brucellosis, of which 7.4% were superficial and 3.7% in the intestine. In dungheap housed flies, 13% had live Brucellosis. From experimentally infected mosquitoes, one can isolate after 48-52 hours Brucellosis in the intestine, and from experimentally infected flies, after 7 days. This is why personnel working in stables are prone to infection. This is one way of contracting the diseases, and even unrelated to the farm, people can thus be contaminated. Likewise we must admit that there are natural habitats where Brucellosis can be found e.g. nature reserves amongst the wildlife. (Wild Boars, Deer, Rabbits and Hares, Foxes, carrion eating birds, insects, etc.). There is in fact a listing of 74 types of wild animals and 53 species of insects that carry Brucellosis. I must here point out that to establish this natural reservoir link that in Germany, there was a 2-year study done in 1964-1965 wherein the researchers tested 7000 animals in 45 nature reserves, and in 19 cases Brucellosis was culturally proven, and in another 102 cases, by serological means. The cultural Brucellosis cases were proven to be from 6 origins, and the serological cases proved to be from 4 different origins. Likewise they proved Brucellosis from bloodsucking insects in 18 cases, and another 20 cases showed the natural tendency towards Brucellosis. A few of the insectal infected animals displayed a long term persistent type of Brucellosis. The most subjective animal to carry the disease was found to be the hare; they were found to be 1.5% of the total contaminated species. McNUTT and PURVIN (1930) found that 1.7% of 5,350 chickens were contaminated by Brucella Abortus Bang and by later research of 10,000 chickens, results proved to be 2% positive. Later there were further studies done in the USA by von GILMAN and BRUNETT, by THOMSEN in Denmark, by von EMMEL in the USA, and by BELLER and STOCKMYER 1933. They concluded that under certain conditions, an infection to humans was possible from fowl to human by slaughtering, as well as from egg to human by eating. To come back to human beings: In the event of a Human mother unwittingly passing on this disease, the second and third generation sufferers are much more difficult to diagnose. Fortunately using Electro acupuncture or B.F.D., this is somewhat easier. However, by comparing symptoms of e.g. Grandmother to Mother to Granddaughter we can often verify the findings, and by treating these various persons with nosode therapy, you notice the change within the body, not only by the homeopath but also by the patient. I was once very privileged to measure with electro-acupuncture 4 generations in the case of another disease which carries toxins to 7 generations and I was amazed at the basic structure of the disease carrying through the generations whilst it became more difficult to distinguish and diagnose per each passing generation. The 94 year old Great-Grandmother actually remembered that her father was the original disease sufferer, so she was not amazed or bewildered by my findings. The youngest recipient of the transmitted genetic toxin was 19 years old, and was already showing many symptoms of the original disease. In other words, Brucella Abortus Bang becomes a genetic disorder, and is transmitted for at least 3 generations; unfortunately, with each generation it becomes harder to identify as it is borne in the new generation congenitally. Case 1:
Case 2:
Two months later I received the results, a) that it was a rare case of Hemochromatosis as she was female. (Apparently it is rare for women to get this disease according to the specialists.) b) that she was too young to normally have it. It apparently occurs more in the 50's or early 60's and c) apart from a liver transplant the specialist could not offer any other advice than draining off 1 pint of blood every week (bloodletting) to try to stop the iron content building up too much in the liver. Further he advised her, because of the unusual circumstances mentioned, to return every three months for a complete blood analysis and eventually a liver transplant. This was the only advice. No medication was given. I had my original suspicion confirmed. I started treating this patient by removing every infection foci I could find, and started her on the enzyme treatment of the company Heel. Coenzyme Compositum, Ubichinon Compositum, and Glyoxal Compositum. As I had also found the first traces of cancer, I included Carcinomium Compositum in the prescription. The results were excellent. After the first 6 weeks I could stop treating her with the Carcinomium Compositum as I could not detect any trace of the cancer. [see The Journal of Degenerative Diseases, Vol. l, Nos 3-4, Cancer and the Mycoplasma, pp. 58-59--Ed.] In subsequent visits I took her off the enzyme treatment as her condition improved. During the entire time the patient continued the bloodletting. Currently she has recently shed her 280th pint. During the enzyme treatment, I came across the Brucella Melitense infection in the Colon transversum, which I had not been able to identify earlier. The patient was not aware of any contamination she had knowingly had herself. However, in her family history, both grandfathers had kept goats. One of these grandfathers had died from an illness which the doctor could not identify. She remembered that he had many fevers of unknown sources before finally passing away. Her brother, two sisters, and a cousin have now been diagnosed as suffering from genetic Hemochromatosis by the specialists. The whole family had a double dose of it as both grandfathers were carriers medically proven by the specialists. Since I have been treating her for the Brucella Melitense there are some definite improvements in her general health although the Hemochromatosis is still there, (possibly for life). Case 3:
This lady openly admitted that they had experienced many problems on the farm for some years. These had taken the form of miscarriages in the sheep: lambs being born too early in term; hand-rearing lambs where their mother had no milk for them, etc. Their veterinary doctor had never looked at these problems, let alone taken appropriate action. This put at risk anyone who drank the ewe's milk or yoghurt, or the cheese made from unpasteurised milk. Thus any humans that ate or drank these products were at risk. So were the next three generations. I have treated this lady farmer for 6 weeks. The results are remarkable. She no longer has any of the M.S.-like symptoms. The liver is being nursed back to health. She feels 500% improved health. The Hypoglycaemic symptoms have all but disappeared, and the symptomatic signs of the Hemochromatosis have halved, and are still lessening. The swollen liver feels smaller and soft as before. It will take possibly another 3 months of treatment. The prognosis is a good one Six weeks later I was able to discharge her from my care. Case 4:
Whilst she was on holiday there she had injured the top of her hand by scratching it open on a gate post. The wound would not heal and eventually had become ulcerated. When she was back home in Southampton she visited her local G.P. and found him to be away, so she saw his locum (replacing G.P.--Ed.). The wound by now had grown a lot larger. The locum doctor asked her if she had anything to do with sheep, to which she replied by telling him of her story of the sheep farm at N. Devon. The locum then treated her hand with a steroid creme, and no more was then done, as the wound had healed. Now, 4 years later, after the healing of the ulcerated hand, I find a Cirrhosis of the liver, and a colon which (along its entire length) has ulcers causing the Cirrhosis. She also had all the classical symptoms of Hemochromatosis. Taking into consideration the condition of the liver, and the state of her general condition I wrote a letter to her G.P. telling him to check his patient's chart, and pointing out to him his locum's findings, and then my diagnosis. I also gave him my most recent diagnosis. At the same time I included an advice prescription as to what I would use to clear up the condition, if he should be inclined to use my method. I gave him a prognosis and possible solutions, and assured him that I would look after our patient. I said I would give him progress reports etc. in the event of his using my advice prescription. I included the name of the importer and the price of the medication. I also told the patient why I was sending her back to her G.P. Also, she did not have the means to buy the medication herself but I assured her that if the G.P. let her down and did not help her, she could always come back to me herself, and I would help her. Case 5:
At present she has a diseased liver, and all the classical markings of Hemochromatosis on her face, her neck, and her chest. She is suffering from a hardening of the liver and has suffered for the past 5 years from pain in the intestines. Six weeks later the lady has returned for her check-up. She has suffered severe reverse vicaration during the first two weeks, pain in the intestine, bloated liver tender to the touch, felt liverish, unable to eat much, pain in pancreas region, joint pains, stiff neck, and frozen shoulders. Upon measuring her body, I found that in about another three weeks the infection will be gone. The liver is more palpable, less swollen, and I could hardly find any infection in the intestine. The skin blotches have all but disappeared, the palms of the hands which used to have a bright red edge around them are now less coloured. The patient reported that she is feeling a lot better; her family has noticed that she has much more energy and that she is no longer in constant pain. All in all, there is much improvement and it appears that the next 6 weeks will more than likely see an end to her problems. Case 6:
The lady had pet goats when she was a child and had a history of illnesses behind her typical of the Brucellosis. She vividly remembered all the symptoms and, as she has a strong medical background, she could accept our findings. At the end of the Brucella Melitensis cure we found her condition to be excellent. During the treatment she clearly felt the reverse vicarations of the illness. By the end of the first 6 weeks, the Sarcoid mark on her skin appeared to be reducing not only in size but also in colour. By the end of the tailing down of the Brucella Melitense treatment it had shrunk to less than 50% of its original size and colouring. [Incidently, Sarcoidosis had very veiled early symptoms, very much like a Brucella Melitensis. I am unable to look into the past, but, from what this medically enlightened patient says, there could be a link in her case between this and the Brucella Melitense. I will keep looking for signs. [See also The Journal of Degenerative Diseases, Vol. 2, #1, pp. 57-67--Ed.] Case 7:
Now that he is on the taildown of the Brucella Melitense treatment, he has let go of his "habit"; his skin colouring has improved drastically; and, he is about to be treated homeopathically to assist him in kicking his smoking habit. In short at this stage I have treated another 28 cases of Brucella Melitense both here (in the U K.) and in Spain and they were nearly all identical in symptoms and results. Save that there was one case in Spain-There was a 34 year old lady who did not take the medication. She suffered many pains as time went on and 18 months later she passed away from liver cancer. By now it must be clear that many women under 40 suffer from this complication of Hemochromatosis which dispels the official thoughts on this converted illness. I now have seen a large number of cases where the enzootic disease of Brucella Melitense which on the face of it had barely recorded links, up to now, with Hemochromatosis. We now see that often it is the infection of Brucella Melitense which is focused in the bowel, that causes iron to be pushed into the liver. This in turn causes the copper coloured blotches on the skin and the redness on the palms of the hands, as well as the breaking down of blood vessels on the face, the neck and the upper chest. We are aware that this enzootic disease of Brucella Melitense is not well recorded, neither are its link-ups to other diseases. Brucella Melitense also displays symptoms of lack of sexual appetite, loss of energy and terrible pains in the Colon, Liver and Pancreas areas. Should my researches be proven to be correct, this Brucella Melitense problem could well take on some of the proportions (hopefully lesser than) of the recent BSE (Mad Cow Disease-Ed.) drama. The British government has up to now never admitted to having a "Brucella Melitense" problem. It is also interesting to note that in 30 years there has never been another "European" investigation into "Brucellosis". This means that in order to research my subject I have had to look at records, some of which are 30 or more years old. This does not mean that in these 30 years past much has changed, apart from governments in Europe no longer reporting on the problem. Another disturbing point of information I came across during my research into this sheep and goat disease is that "unfit for human consumption" milk powder, due to its expiry date having lapsed by a great margin, is now being fed to ewes which have reached their economical end of breeding. The ewes really go for this supplement and as a result, apparently the colour of the mutton changes to that of a lamb, so that dishonest brokers can get the lamb price for old, artificially coloured mutton. I hope that my empirically based findings will assist a scientist in taking a deeper look into the problems regarding Brucella Melitense. Conclusions
2) If further research proves my theory that Hemochromatosis is indeed caused by Brucella Melitense, and that if the disease of Hemochromatosis is found at a fairly early stage, by treating it in the tradition of Homotoxicology the disease can be stopped. This would mean that future generations stemming from the patient need not suffer from the consequences of the disease or its eventual secondary liver disease. 3) There is a definite need for the source of the disease to be eradicated by the abrupt stopping of the farmer from selling raw milk products, (like milk, cream, butter and farm made cheese and yoghurt.). I have just cured myself from Brucella Abortus Bang, which I contracted from cheese made in Britain sold under the brand label of one of the largest Superstores in this country. Obviously the ministry of Health allows these Superstores to purchase direct from farmers, unpasteurised milk products such as cream, farm cheese, yoghurts, etc. which are then sold in the Superstores under their own brands. Perhaps it would be advantageous to adopt the French system where the seller of this produce has to display a certificate assuring that the produce on display is made from "Brucellosis Free" animals and is signed by the local veterinarian, as well as the State Veterinarian. In France this test must be made every six months otherwise there is NO sale of goods. It may also be wise to adopt the Dutch method of dealing with an outbreak. Thus if one animal on the farm is positive, then the State Veterinarians move in culling every single animal on that farm, burning all carcases, burning all sheds etc .where contamination from the animals could be found. It could also be suggested that the land should be proclaimed as non-grazing land until no further contamination is found there by the State Veterinary department or an independent responsible agency. 4) As there has never been a great investigation done on sheep and goat's diseases at any level, and in the light of what I uncovered in the form of malpractices in feeding aged sheep milk powder "not fit for human consumption" to change the colour of the meat so that it can fetch the higher Lamb price from the consumer, I would like to believe that perhaps this orignial paper can stir scientists and perhaps even governments into taking some action. 5) If my assumptions and conclusions are correct and verified by the 29 out of the 35 cases I have studied, I may safely deduce that Brucella Melitense has indeed an influence on complications of iron reflux going into the liver as a result of intestinal infection. This in turn may cause Cirrhosis and Hemochromatosis-like symptoms. I hope that by my research and empirical study I have opened the way to a far more in-depth research into Brucella Melitense and Hemochromatosis. I hope some relief may to given to the thousands of people suffering from this affliction. (According to a "specialist" at the St. James Hospital in Leeds, the number is 300 patients per million.) 6) All my findings in this original paper were done empirically and any medications used on the patients mentioned were biological or homeopathic. I am open to criticism or correction by any researcher doing an in-depth study as to further findings in the subject of Brucella Melitense and Hemochromatosis. Lastly
Upon measuring her with the BFD I found that the original disease was Brucella Melitense. According to the strength of the ampoules required it pointed out exactly 17 years. I found it in her liver, spleen, stomach, pancreas, intestines. She is now going to bring her son and husband. Upon further investigation it turned out that these houses were built on the ex-dairy farm of Farmer Frampton. This family has by now died out but before this township was built it was known that a) Farmer Frampton used these fields for mixed herds of cattle and sheep and goats. b) It is also known to some of the local elderly residents that Farmer Frampton's mixed herds had many
The government has just had veterinarians doing blood tests in Devon checking up on this disease. They took some 20 sheep at random out of the thousands of sheep at each farm, took some blood off them, let the blood cool down and the next day these blood vials were sent of to the laboratories to be tested. Now what is wrong is that Brucellosis
can only be cultured in body temperature blood, so if the body temperature
is I have just seen a sheep dairy farmer whom I have checked regularly before, and have not before found anything such as Brucella Melitense or Brucella Abortus Bang. Now he has installed a cheese making plant and has started purchasing sheep milk from 7 other farmers. He now has Brucella Melitense in his own body from the milk he has purchased and not pasteurised before making sheep yoghurt and sheep cheese. In doing tests with the milk of his producers I found both Brucella Melitense and Brucella Abortus Bang in the milk samples supplied to me by the farmer/cheese producer. It makes me wonder how the government agency allows unpasteurised dairy products to be sold to the public. The other startling discovery is that the government here allows the dairy boards to manipulate the temperatures of the pasteurisation process of milk, viz in order to save energy in the pasteurisation process they allow 63 degrees Centigrade as the maximum and zero degrees as the lower temperatures. As a result I have actually isolated Brucella Abortus Bang, Brucella Melitensis, Tuberculinum Bovinum, Tuberculinum De Nijs, Mycoplasma Bovinum, Mycoplasma Dispar and Mycoplasma Bovis and Mycoplasma Ovine in milk taken off the shelves at the Supermarkets. In effect the pasteurised milk is highly dangerous and nobody wants to listen. This is of course besides the contamination the population is placed under by Germ Warfare experimentation. The Ministry of Defence secret plant has admitted quite recently that they had done 153 tests of this nature upon the population, a lot of this was with Brucellosis, and Mycoplasma infections. Brucella
Suis PROPHYLAXIS
Wipe out Brucella infections in cattle (either by nosode means or wipe out all animals on contaminated farms). Never eat raw or partially cooked meat, or unpasteurized milk products, and never drink unpasteurized milk. (Unfortunately a strong Brucellosis infection will even stand being Pasteurised. Admittedly it is rare, but it can sometimes survive it.) Most Cheddar Cheeses in Canada and the U.K. are farmhouse cheeses and are made from unpasteurised milk so avoid eating unprocessed cheeses. THERAPY Bibliography: TAYLOR, R. M., LISBONNE, M:, VIDAL, L. F. and HAZEMANN, R. H.: Investigation on Undulant Fever in France; 1938 from the research centre on Undulant fever institute BuissonBertrand, Montpelier, France: League of Nations Bulletin of the Health Organization, Vol. 7, p. 503, June. SANDER, J. F.: Undulant Fever in Children: 1933: Journal of Michigan State Medical Society, Vol. 32, p. 109. KRISTENSEN, M., HELMS, T. and MARTENSSON: Serologic Researches in Bacillus Abortus infections: 1931, Ugeskrift for Laeger, vol. 93,p.51. ROGER, H., and POURSINES, Y.: The Meningeo-Neuro-Brucellosis, 1938, Paris-Masson et Cie. K. C. CORLOFF: Malta Fever and the Eye: 1928, Klinische Monatblatter fur Augenheilkunde vol. 81,p.252. LUNDSGAARD, K. K. K.: A Case of Iridocyclitis in association with Febris Undulans Nostra, 1928, Acta Opthalmologica, vol. 6, p. 1. JONES, L. W., and NORRIS, J. L.: Undulant Fever with Visual Disturbances: 1941, New York State Journal of Medicine, Vol. P. 17625. Brucelloseforschung", 1965, republished J. Parnas, a monographic work, by 10 authors from Poland, Russia, Chechoslovakia, East Germany. DR. HUDDLESTON: Brucella Infections in Animals and Man, 1944, published by the Commonwealth Fund. PARNAS, J., KRUGER, W., and TOPPICH, E.: Die Brucellose des Menschen, republished Berlin, 1966, VEB Verlag Volk und Gesundheit. LINDEMAN, M. G. Brucella Abortus Bang, 1990, a paper delivered to the Steigerwalt symposium. LINDEMAN, A. J.: Brucella Melitense and its relationship to diseases of the intestines and liver, in particular, its relationship to Haemochromatosis, submitted May 1966 to the Internationale Gesellshaft fur Homotoxocologie und Homotoxische Therapie, Baden Baden. About the
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